Background: Tachycardiomyopathy is an illness that is caused by accelerated and/or irregular cardiac rhythm, which result in myocardial damage and heart failure. There are no clear criteria for diagnosing this condition, but patient have weakness of the heart muscle with presence of some forms of heart rhythm. Non-compaction cardiomyopathy is disease which is result of a disturbance in the early phase of myocardial left ventricular development with deep recessions between trabeculae and abnormal trabeculation/hyperproliferation. This disease is associated with left ventricular failure, cardiac arrhythmias, embolic events, and sudden cardiac death. (1, 2)

Case report: 39-year-old man with history of palpitation, ventricular arrhythmias, now complaining of tachycardia, palpitation and fatigue, was hospitalized in our Hospital for treatment of episode of paroxysmal supraventricular tachycardia which was converted in sinus rhythm with adenosine. Patient was not treated with any medical therapy before. Electrocardiography was shown episode of supraventricular tachycardia (PSVT) with short RP interval which was encouraged with ventricular extrasystole with retrograde conduction. We were suspected at atrioventricular circular tachycardia with retrograde conduction with aberrant beam. Echocardiography detected non-compaction cardiomyopathy with low left ventricular ejection fraction (LVEF), without element of coronary artery disease. Considering the earliest echocardiography findings with preserved LVEF, we conclude that with the existence of non-compaction cardiomyopathy, the tachycardiomyopathy was followed up to an additional reduced LVEF. With administered drug therapy followed by a significant reduction in the number of episodes of supraventicular tachycardia, the patient was discharged with the recommendation of taking verapamil, sacubitril/valsartan, eplerenone, dabigatran and we recommended ablation. After ablation, echocardiography findings that has been done, have shown global hypocontractility with preserved contractility posterolateral and better LVEF. After three months of ablation we made control magnetic resonance wich has shown discretely dimension reduction of left ventricle and still non-compaction cardiomyopathy with global hypokinesia but discreet recovery of LVEF and better feeling of patient.

Conclusions: The current clinical case have shown that the tachycardia weakened the global LVEF in this young man, who also have non-compaction cardiomyopathy, which was slightly improvement when we manage the fast tachycardia with drugs and ablation.